Original Contribution Interactions Between Genome-wide Significant Genetic Variants and Circulating Concentrations of Insulin-like Growth Factor 1, Sex Hormones, and Binding Proteins in Relation to Prostate Cancer Risk in the National Cancer Institute Breast and Prostate Cancer Cohort Consortium

نویسندگان

  • Konstantinos K. Tsilidis
  • Ruth C. Travis
  • Paul N. Appleby
  • Naomi E. Allen
  • Sara Lindstrom
  • Fredrick R. Schumacher
  • David Cox
  • Ann W. Hsing
  • Jing Ma
  • Gianluca Severi
  • Demetrius Albanes
  • Jarmo Virtamo
  • Heiner Boeing
  • H. Bas Bueno-de-Mesquita
  • Mattias Johansson
  • J. Ramón Quirós
  • Elio Riboli
  • Afshan Siddiq
  • Anne Tjønneland
  • Dimitrios Trichopoulos
  • Rosario Tumino
  • J. Michael Gaziano
  • Edward Giovannucci
  • David J. Hunter
  • Peter Kraft
  • Meir J. Stampfer
  • Graham G. Giles
  • Gerald L. Andriole
  • Sonja I. Berndt
  • Stephen J. Chanock
  • Richard B. Hayes
  • Timothy J. Key
چکیده

Konstantinos K. Tsilidis*, Ruth C. Travis, Paul N. Appleby, Naomi E. Allen, Sara Lindstrom, Fredrick R. Schumacher, David Cox, Ann W. Hsing, Jing Ma, Gianluca Severi, Demetrius Albanes, Jarmo Virtamo, Heiner Boeing, H. Bas Bueno-de-Mesquita, Mattias Johansson, J. Ramón Quirós, Elio Riboli, Afshan Siddiq, Anne Tjønneland, Dimitrios Trichopoulos, Rosario Tumino, J. Michael Gaziano, Edward Giovannucci, David J. Hunter, Peter Kraft, Meir J. Stampfer, Graham G. Giles, Gerald L. Andriole, Sonja I. Berndt, Stephen J. Chanock, Richard B. Hayes, and Timothy J. Key

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The insulin-like growth factor I receptor (IGF-IR) plays an essential role in the establishment and maintenance of transformed phenotype. Interference with the IGF-IR pathway by antisense causes reversal of the transformed phenotype in many rodent and human tumor cell lines. We stably transfected the PC-3 human prostate cancer cell line with an IGF-IR antisense RNA expression plasmid. The ...

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تاریخ انتشار 2012